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Research in the Sanderson lab is focused on understanding brain damage caused by cardiac arrest, ischemic stroke, neonatal hypoxia/ischemia, and traumatic brain injury. Two primary avenues of investigation are (1) the role mitochondrial dysfunction in death of neurons during post-ischemic reperfusion and (2) the development and clinical translation of a neuroprotective therapies that reduce neurological injury.
Research in the Sanderson lab is focused on understanding brain damage caused by cardiac arrest, ischemic stroke, neonatal hypoxia/ischemia, and traumatic brain injury. Two primary avenues of investigation are (1) the role mitochondrial dysfunction in death of neurons during post-ischemic reperfusion and (2) the development and clinical translation of a neuroprotective therapies that reduce neurological injury.
Our work is made possible through the support of our partners, sponsors, and funding agencies:
Our work is made possible through the support of our partners, sponsors, and funding agencies:
Part of:
Part of:
Dept. of Molecular and Integrative Physiology
Neuroscience Graduate Program (NGP)
Max Harry Weil Institute for Critical Care Research and Innovation
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